To elucidate the mechanism of renal injury following intestinal ischemia/reperfusion, reactive oxygen metabolites in kidney and plasma were examined in 20 rats following intestinal ischemia/reperfusion by measurement of lipid proxidation (LP).The plasma lipid peroxide concentration after reperfusion was higher than that of the contol group (Ｐ＜0.01),the LP in kidney homogenate was also significantly higher (Ｐ＜0.01) following intestinal ischemia/reperfusion.Our study suggests that reactive oxygen metabolites after intestinal ischemia/reperfusion plays an important role in kidney injury.
Citation：
Zou Yang,Lin Qingtian.. KIDNEY INJURY AFTER INTESTINAL ISCHEMIA/REPERFUSION IN RATS. CHINESE JOURNAL OF BASES AND CLINICS IN GENERAL SURGERY, 1999, 6(1): 11-12. doi:
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- 1. Yagi K. Assay for blood plasma or serum lipid peroxides.Methods Enzymol, 1984; 105(4)∶328.
- 2. Caty MG, Schmeling DJ, Freid HP, et al. A promoter of xanthire oxidase activity in intestinal ischemia/reperfusion. J Pediatr Surg, 1990; 25(2)∶218.
- 3. Schoenberg MH, Beger HG. Reperfusion injury after intestinal ischemia. Critical Care Medicine, 1993; 21(9)∶1376.
- 4. Schoenberg MH, Fredholm BB, Haglund U, et al. Studies on the oxygen radical mechanism involved in the small intestinal reperfusion damage. Acta Physiol Scand, 1985; 124(4)∶581.
